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  4.        <title>Nature Reviews Immunology</title>
  5.        <description>Immunology is a diverse and growing discipline that can be defined as the study of the tissues, cells and molecules involved in host defence mechanisms, how the body defends itself against disease, and what happens when it all goes wrong. Nature Reviews Immunology provides in-depth coverage of this field, from fundamental mechanisms to translational aspects of basic research, and reviews the field’s most important developments. All Review and Perspective articles are carefully commissioned by the editors and written by leaders in the field. Articles are subject to rigorous peer review and provide high-quality and authoritative coverage of the field in each issue. Articles are carefully tailored by the editors to provide accessible information for non-specialists, and this is additionally enhanced with the use of Glossary terms and highlighted references. Each issue also contains Research Highlight articles – short pieces written by the editors that summarize the results from recent hot research papers.</description>
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  41.        <title>Nature Reviews Immunology</title>
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  47.            <title><![CDATA[Opportunities and challenges for T cell-based influenza vaccines]]></title>
  48.            <link>https://www.nature.com/articles/s41577-024-01030-8</link>
  49.            <content:encoded>
  50.                <![CDATA[<p>Nature Reviews Immunology, Published online: 02 May 2024; <a href="https://www.nature.com/articles/s41577-024-01030-8">doi:10.1038/s41577-024-01030-8</a></p>Compared with many other vaccines, current vaccines against influenza provide only limited protection. Here, the authors describe the challenges and recent attempts at generating T cell-based vaccines. It may be important to combine T cell-based vaccines with antibody-based vaccines to provide long-lasting immunity across influenza virus strains.]]></content:encoded>
  51.            <dc:title><![CDATA[Opportunities and challenges for T cell-based influenza vaccines]]></dc:title>
  52.            <dc:creator>Tim R. Mosmann</dc:creator><dc:creator>Andrew J. McMichael</dc:creator><dc:creator>Alexandre LeVert</dc:creator><dc:creator>John W. McCauley</dc:creator><dc:creator>Jeffrey W. Almond</dc:creator>
  53.            <dc:identifier>doi:10.1038/s41577-024-01030-8</dc:identifier>
  54.            <dc:source>Nature Reviews Immunology, Published online: 2024-05-02; | doi:10.1038/s41577-024-01030-8</dc:source>
  55.            <dc:date>2024-05-02</dc:date>
  56.            <prism:publicationName>Nature Reviews Immunology</prism:publicationName>
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  62.            <title><![CDATA[Defining the balance between optimal immunity and immunopathology in influenza virus infection]]></title>
  63.            <link>https://www.nature.com/articles/s41577-024-01029-1</link>
  64.            <content:encoded>
  65.                <![CDATA[<p>Nature Reviews Immunology, Published online: 02 May 2024; <a href="https://www.nature.com/articles/s41577-024-01029-1">doi:10.1038/s41577-024-01029-1</a></p>An optimal immune response to influenza virus strikes a balance between protective antiviral immune mechanisms and detrimental immunopathology. Here, the authors review the immune mechanisms responsible for each side of this balance and how this may inform future vaccine design.]]></content:encoded>
  66.            <dc:title><![CDATA[Defining the balance between optimal immunity and immunopathology in influenza virus infection]]></dc:title>
  67.            <dc:creator>Thi H. O. Nguyen</dc:creator><dc:creator>Louise C. Rowntree</dc:creator><dc:creator>Brendon Y. Chua</dc:creator><dc:creator>Ryan S. Thwaites</dc:creator><dc:creator>Katherine Kedzierska</dc:creator>
  68.            <dc:identifier>doi:10.1038/s41577-024-01029-1</dc:identifier>
  69.            <dc:source>Nature Reviews Immunology, Published online: 2024-05-02; | doi:10.1038/s41577-024-01029-1</dc:source>
  70.            <dc:date>2024-05-02</dc:date>
  71.            <prism:publicationName>Nature Reviews Immunology</prism:publicationName>
  72.            <prism:doi>10.1038/s41577-024-01029-1</prism:doi>
  73.            <prism:url>https://www.nature.com/articles/s41577-024-01029-1</prism:url>
  74.        </item>
  75.    
  76.        <item rdf:about="https://www.nature.com/articles/s41577-024-01027-3">
  77.            <title><![CDATA[DAMP sensing and sterile inflammation: intracellular, intercellular and inter-organ pathways]]></title>
  78.            <link>https://www.nature.com/articles/s41577-024-01027-3</link>
  79.            <content:encoded>
  80.                <![CDATA[<p>Nature Reviews Immunology, Published online: 29 April 2024; <a href="https://www.nature.com/articles/s41577-024-01027-3">doi:10.1038/s41577-024-01027-3</a></p>Here, Rongbin Zhou and colleagues review the different types of damage-associated molecular pattern (DAMP) that trigger sterile inflammation via pattern recognition receptors. The authors group these DAMPs on the basis of whether they arise from inside cells, from neighbouring cells or from distant tissues, and they discuss the relevance of such DAMPs in various inflammatory disease settings.]]></content:encoded>
  81.            <dc:title><![CDATA[DAMP sensing and sterile inflammation: intracellular, intercellular and inter-organ pathways]]></dc:title>
  82.            <dc:creator>Yi Huang</dc:creator><dc:creator>Wei Jiang</dc:creator><dc:creator>Rongbin Zhou</dc:creator>
  83.            <dc:identifier>doi:10.1038/s41577-024-01027-3</dc:identifier>
  84.            <dc:source>Nature Reviews Immunology, Published online: 2024-04-29; | doi:10.1038/s41577-024-01027-3</dc:source>
  85.            <dc:date>2024-04-29</dc:date>
  86.            <prism:publicationName>Nature Reviews Immunology</prism:publicationName>
  87.            <prism:doi>10.1038/s41577-024-01027-3</prism:doi>
  88.            <prism:url>https://www.nature.com/articles/s41577-024-01027-3</prism:url>
  89.        </item>
  90.    
  91.        <item rdf:about="https://www.nature.com/articles/s41577-024-01039-z">
  92.            <title><![CDATA[Noncanonical inflammasome mediates BBB breakdown]]></title>
  93.            <link>https://www.nature.com/articles/s41577-024-01039-z</link>
  94.            <content:encoded>
  95.                <![CDATA[<p>Nature Reviews Immunology, Published online: 26 April 2024; <a href="https://www.nature.com/articles/s41577-024-01039-z">doi:10.1038/s41577-024-01039-z</a></p>Wei et al. report a role for membrane perforation mediated by gasdermin D pores in disruption of the blood–brain barrier.]]></content:encoded>
  96.            <dc:title><![CDATA[Noncanonical inflammasome mediates BBB breakdown]]></dc:title>
  97.            <dc:creator>Kirsty Minton</dc:creator>
  98.            <dc:identifier>doi:10.1038/s41577-024-01039-z</dc:identifier>
  99.            <dc:source>Nature Reviews Immunology, Published online: 2024-04-26; | doi:10.1038/s41577-024-01039-z</dc:source>
  100.            <dc:date>2024-04-26</dc:date>
  101.            <prism:publicationName>Nature Reviews Immunology</prism:publicationName>
  102.            <prism:doi>10.1038/s41577-024-01039-z</prism:doi>
  103.            <prism:url>https://www.nature.com/articles/s41577-024-01039-z</prism:url>
  104.        </item>
  105.    
  106.        <item rdf:about="https://www.nature.com/articles/s41577-024-01038-0">
  107.            <title><![CDATA[FAS signalling pathway is crucial for CAR T cell persistence]]></title>
  108.            <link>https://www.nature.com/articles/s41577-024-01038-0</link>
  109.            <content:encoded>
  110.                <![CDATA[<p>Nature Reviews Immunology, Published online: 26 April 2024; <a href="https://www.nature.com/articles/s41577-024-01038-0">doi:10.1038/s41577-024-01038-0</a></p>A preprint by Yi et al. shows a role for the FAS signalling pathway in controlling the persistence of CAR-modified lymphocytes.]]></content:encoded>
  111.            <dc:title><![CDATA[FAS signalling pathway is crucial for CAR T cell persistence]]></dc:title>
  112.            <dc:creator>Robyn Loves</dc:creator><dc:creator>Eyal Grunebaum</dc:creator>
  113.            <dc:identifier>doi:10.1038/s41577-024-01038-0</dc:identifier>
  114.            <dc:source>Nature Reviews Immunology, Published online: 2024-04-26; | doi:10.1038/s41577-024-01038-0</dc:source>
  115.            <dc:date>2024-04-26</dc:date>
  116.            <prism:publicationName>Nature Reviews Immunology</prism:publicationName>
  117.            <prism:doi>10.1038/s41577-024-01038-0</prism:doi>
  118.            <prism:url>https://www.nature.com/articles/s41577-024-01038-0</prism:url>
  119.        </item>
  120.    
  121.        <item rdf:about="https://www.nature.com/articles/s41577-024-01037-1">
  122.            <title><![CDATA[Harnessing our lived experience for science communication]]></title>
  123.            <link>https://www.nature.com/articles/s41577-024-01037-1</link>
  124.            <content:encoded>
  125.                <![CDATA[<p>Nature Reviews Immunology, Published online: 24 April 2024; <a href="https://www.nature.com/articles/s41577-024-01037-1">doi:10.1038/s41577-024-01037-1</a></p>Adrian Liston, professor of pathology at the University of Cambridge, UK, has published several illustrated children’s books on the topic of vaccination and has developed a computer game called ‘VirusFighter’. Here, he shares his thoughts on how to become an effective science communicator.]]></content:encoded>
  126.            <dc:title><![CDATA[Harnessing our lived experience for science communication]]></dc:title>
  127.            <dc:creator>Adrian Liston</dc:creator>
  128.            <dc:identifier>doi:10.1038/s41577-024-01037-1</dc:identifier>
  129.            <dc:source>Nature Reviews Immunology, Published online: 2024-04-24; | doi:10.1038/s41577-024-01037-1</dc:source>
  130.            <dc:date>2024-04-24</dc:date>
  131.            <prism:publicationName>Nature Reviews Immunology</prism:publicationName>
  132.            <prism:doi>10.1038/s41577-024-01037-1</prism:doi>
  133.            <prism:url>https://www.nature.com/articles/s41577-024-01037-1</prism:url>
  134.        </item>
  135.    
  136.        <item rdf:about="https://www.nature.com/articles/s41577-024-01036-2">
  137.            <title><![CDATA[Weaker skin immunity in males due to androgen effects on ILC2s]]></title>
  138.            <link>https://www.nature.com/articles/s41577-024-01036-2</link>
  139.            <content:encoded>
  140.                <![CDATA[<p>Nature Reviews Immunology, Published online: 24 April 2024; <a href="https://www.nature.com/articles/s41577-024-01036-2">doi:10.1038/s41577-024-01036-2</a></p>Sex hormones in male mice negatively regulate type 2 innate lymphoid cells in the skin, impairing the induction and activation of dendritic cells and thereby contributing to differences in immunity in males and females.]]></content:encoded>
  141.            <dc:title><![CDATA[Weaker skin immunity in males due to androgen effects on ILC2s]]></dc:title>
  142.            <dc:creator>Lucy Bird</dc:creator>
  143.            <dc:identifier>doi:10.1038/s41577-024-01036-2</dc:identifier>
  144.            <dc:source>Nature Reviews Immunology, Published online: 2024-04-24; | doi:10.1038/s41577-024-01036-2</dc:source>
  145.            <dc:date>2024-04-24</dc:date>
  146.            <prism:publicationName>Nature Reviews Immunology</prism:publicationName>
  147.            <prism:doi>10.1038/s41577-024-01036-2</prism:doi>
  148.            <prism:url>https://www.nature.com/articles/s41577-024-01036-2</prism:url>
  149.        </item>
  150.    
  151.        <item rdf:about="https://www.nature.com/articles/s41577-024-01022-8">
  152.            <title><![CDATA[Engineering immune-evasive allogeneic cellular immunotherapies]]></title>
  153.            <link>https://www.nature.com/articles/s41577-024-01022-8</link>
  154.            <content:encoded>
  155.                <![CDATA[<p>Nature Reviews Immunology, Published online: 24 April 2024; <a href="https://www.nature.com/articles/s41577-024-01022-8">doi:10.1038/s41577-024-01022-8</a></p>Genome editing approaches can be used to confer immune-evasive properties to allogeneic cellular immunotherapies, with the aim of achieving persistent responses and efficiencies that are comparable to those of autologous chimeric antigen receptor T cell therapies. This Perspective discusses how current knowledge about viral or tumour immune evasion could be incorporated into the design of off-the-shelf tumour-specific T and NK cells for the production of cost-effective and scalable cancer immunotherapies.]]></content:encoded>
  156.            <dc:title><![CDATA[Engineering immune-evasive allogeneic cellular immunotherapies]]></dc:title>
  157.            <dc:creator>Karen E. Martin</dc:creator><dc:creator>Quirin Hammer</dc:creator><dc:creator>Karlo Perica</dc:creator><dc:creator>Michel Sadelain</dc:creator><dc:creator>Karl-Johan Malmberg</dc:creator>
  158.            <dc:identifier>doi:10.1038/s41577-024-01022-8</dc:identifier>
  159.            <dc:source>Nature Reviews Immunology, Published online: 2024-04-24; | doi:10.1038/s41577-024-01022-8</dc:source>
  160.            <dc:date>2024-04-24</dc:date>
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